Search results for "Slice preparation"

showing 10 items of 12 documents

Changes in Cerebral Amino Acid Transport During Development

1976

The transport of metabolites to and from the central nervous system is of considerable interest. To a greater extent than most other tissues, central nervous system tissue invitro takes up amino acids to well above their concentrations in the incubation medium. Presumably the transport systems responsible for this uptake and for efflux invitro are also those responsible for transport between brain cells in living animals2.

Citric acid cyclechemistry.chemical_classificationmedicine.anatomical_structureSlice preparationBiochemistryChemistryCentral nervous systemmedicineEffluxIncubationIn vitroFetal brainAmino acid
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Phospholipid breakdown and choline release under hypoxic conditions: inhibition by bilobalide, a constituent of Ginkgo biloba

1997

A marked increase of choline release from rat hippocampal slices was observed when the slices were superfused with oxygen-free buffer, indicating hypoxia-induced hydrolysis of choline-containing phospholipids. This increase of choline release was suppressed by bilobalide, an ingredient of Ginkgo biloba, but not by a mixture of ginkgolides. The EC50 value for bilobalide was 0.38 microM. In ex vivo experiments, bilobalide also inhibited hypoxia-induced choline release when given p.o. in doses of 2-20 mg/kg 1 h prior to slice preparation. The half-maximum effect was observed with 6 mg/kg bilobalide. A similar effect was noted after p.o. administration of 200 mg/kg EGb 761, a ginkgo extract con…

MaleDrug Evaluation PreclinicalCyclopentanesPharmacologyHippocampusCholinechemistry.chemical_compoundSlice preparationBilobalideAnimalsCholineRats WistarGinkgolidesFuransHypoxia BrainMolecular BiologyPhospholipidsEC50biologyPlant ExtractsGinkgo bilobaGeneral NeuroscienceGinkgobiology.organism_classificationRatsPlant LeavesGinkgolidesLogistic ModelschemistryBiochemistryNeurology (clinical)DiterpenesEx vivoDevelopmental BiologyBrain Research
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The superoxide anion is involved in the induction of long-term potentiation in the rat somatosensory cortex in vitro.

2004

Abstract The involvement of the superoxide anion (O2−) in the induction of neocortical long-term potentiation (LTP) was examined in rat brain slices containing the primary somatosensory cortex. Field potentials evoked by stimulation in cortical layer IV were recorded from layer II/III. In control experiments, tetanic high-frequency stimulation (HFS) resulted in essentially input-specific, NMDA receptor-dependent LTP (20.2±3.0% increase in field potential amplitude). When the availability of intracellular O2− was reduced by application of the cell membrane-permeable O2− scavengers MnTBAP or CP-H (spin trap), HFS-induced LTP was attenuated to 12.0±1.7% and 8.7±3.1% increase, respectively. In …

MaleLong-Term PotentiationStimulationNeurotransmissionBiologyIn Vitro TechniquesSuperoxide dismutaseRats Sprague-Dawleychemistry.chemical_compoundSlice preparationSuperoxidesAnimalsMolecular BiologySuperoxideGeneral NeuroscienceLong-term potentiationSomatosensory CortexRatschemistryBiophysicsbiology.proteinNMDA receptorNeurology (clinical)NeuroscienceIntracellularDevelopmental BiologyBrain research
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Optical release of caged glutamate for stimulation of neurons in the in vitro slice preparation

2005

Optical stimulation techniques prove useful to map func- tional inputs in the in vitro brain slice preparation: Glutamate released by a focused beam of UV light induces action potentials, which can be detected in postsynaptic neurons. The direct activation effect is influenced by factors such as compound concentration, focus depth, light absorption in the tissue, and sensitivity of different neuronal do- mains. We analyze information derived from direct stimulation ex- periments in slices from rat barrel cortex and construct a computa- tional model of a layer V pyramidal neuron that reproduces the experimental findings. The model predictions concerning the influ- ence of focus depth on inpu…

MalePatch-Clamp TechniquesUltraviolet RaysModels NeurologicalBiomedical EngineeringAction PotentialsStimulationIn Vitro TechniquesCaged glutamateBrain mappingBiomaterialsOpticsSlice preparationGlutamatesPostsynaptic potentialmedicineAnimalsComputer SimulationRats WistarMicroscopy VideoPhotolysisbusiness.industryChemistryPyramidal CellsGlutamate receptorEquipment DesignSomatosensory CortexBarrel cortexAtomic and Molecular Physics and OpticsRatsElectronic Optical and Magnetic Materialsmedicine.anatomical_structureLens (anatomy)SynapsesBiophysicsbusinessJournal of Biomedical Optics
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Subthreshold oscillation of the membrane potential in magnocellular neurones of the rat supraoptic nucleus

2000

The hypothalamic supraoptic nucleus (SON) contains two major populations of magnocellular neurosecretory neurones, producing and secreting vasopressin and oxytocin, respectively (for review see Poulain & Wakerley 1982). Neurones of a subpopulation of supraoptic neurosecretory cells share the capability of generating phasic bursts of action potentials. In these neurones, action potentials are succeeded by a depolarizing afterpotential (DAP; Andrew, 1987; Armstrong et al. 1994; Li et al. 1995). Depending on the discharge frequency, DAPs summate, eventually resulting in the generation of a plateau potential that gives rise to the discharge of a long-lasting train of action potentials. Thus, DA…

MalePhysiologyTetrodotoxinCholinergic AgonistsIn Vitro TechniquesSupraoptic nucleusMembrane PotentialsRats Sprague-DawleyBurstingSlice preparationBiological ClocksOscillometryPotassium Channel BlockersmedicineAnimalsPremovement neuronal activityMagnesiumAnesthetics LocalNeuronsMembrane potentialNeocortexChemistrymusculoskeletal neural and ocular physiologySodium channelTetraethylammoniumDepolarizationOriginal ArticlesRatsmedicine.anatomical_structurenervous systemCalciumSupraoptic NucleusNeuroscienceHeptanolProcaineCadmiumThe Journal of Physiology
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Components of after-hyperpolarization in magnocellular neurones of the rat supraoptic nucleusin vitro

1998

1. The pharmacological sensitivity of hyperpolarizing components of spike train after-potentials was examined in sixty-one magnocellular neurones of the rat supraoptic nucleus using intracellular recording techniques in a brain slice preparation. 2. In 26 % of all neurones a slow after-hyperpolarization (AHP) was observed in addition to a fast AHP. In 31 % of all neurones a depolarizing after-potential (DAP) was observed. 3. The fast AHP was blocked by apamin whereas the slow AHP was blocked by charybdotoxin (ChTX). The DAP was enhanced by ChTX or a DAP was unmasked if not present during the control period. 4. Low concentrations of TEA (0.15-1.5 mM) induced effects on the slow AHP and the D…

MalePotassium ChannelsCharybdotoxinPhysiologySpike trainAction PotentialsApaminSupraoptic nucleusRats Sprague-DawleySK channelchemistry.chemical_compoundSlice preparationAnimalsNeuronsKv1.3 Potassium ChannelVoltage-gated ion channelChemistryMargatoxinTetraethylammoniumOriginal ArticlesIberiotoxinImmunohistochemistryRatsElectrophysiologyApaminPotassium Channels Voltage-GatedBiophysicsSupraoptic NucleusNeuroscienceThe Journal of Physiology
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Increased Hypoxic Tolerance by Chemical Inhibition of Oxidative Phosphorylation: “Chemical Preconditioning”

1997

A short ischemic episode preceding sustained ischemia is known to increase tolerance against ischemic cell death. We report early-onset long-lasting neuroprotection against in vitro hypoxia by preceding selective chemical inhibition of oxidative phosphorylation: “chemical preconditioning.” The amplitude of CA1population spikes (psap) in hippocampal slices prepared from control animals (control slices) was 31 ± 27% (mean ± SD) upon 45-min recovery from 15-min in vitro hypoxia. In slices prepared from animals treated in vivo with 20 mg/kg 3-nitropropionate (3-np) 1–24 h prior to slice preparation (preconditioned slices), psap improved to 90 ± 15% (p < 0.01). Posthypoxic oxygen free radical…

MalePotassium ChannelsFree RadicalsPopulationIschemiaNerve Tissue ProteinsBiologyPharmacologyHippocampusNeuroprotectionOxidative PhosphorylationBrain Ischemia030218 nuclear medicine & medical imagingGlibenclamide03 medical and health sciencesAdenosine Triphosphate0302 clinical medicineSlice preparationIn vivoGlyburidemedicineAnimalsEnzyme InhibitorsRats WistarHypoxia BraineducationNeuronseducation.field_of_studyAntagonistHypoxia (medical)NADNitro Compoundsmedicine.diseaseCell HypoxiaRatsSuccinate DehydrogenaseNeuroprotective AgentsNeurologyAnesthesiaNeurology (clinical)Propionatesmedicine.symptomReactive Oxygen SpeciesCardiology and Cardiovascular Medicine030217 neurology & neurosurgerymedicine.drugJournal of Cerebral Blood Flow & Metabolism
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Comparative investigations on the actions of ACTH1-24, somatostatin, neurotensin, substance P and vasopressin on locus coeruleus neuronal activity in…

1987

A considerable number of neuropeptides have been localized immunohistochemically in the area of the locus coeruleus of the rat. The objective of this study was to assess the actions of some of these transmitter candidates on spontaneously active locus coeruleus neurons in vitro. The effects of bath-applied peptides on the discharge rate of individual locus coeruleus neurons were investigated. A midpontine slice preparation of the gerbil brain was used. Excitatory dose-dependent effects were found with four peptides with the following rank order of potency: Substance P, (Arg8)-vasopressin, neurotensin, ACTH1–24. Somatostatin hyperpolarized all neurons tested. Given the pronounced effects see…

Maleendocrine systemmedicine.medical_specialtyVasopressinVasopressinsNeuropeptideSubstance PIn Vitro TechniquesSubstance Pchemistry.chemical_compoundSlice preparationInternal medicinemedicinePremovement neuronal activityAnimalsNeurotensinPharmacologyNeuronsChemistryNeuropeptidesGeneral MedicineArginine VasopressinEndocrinologySomatostatinnervous systemLocus coeruleusCosyntropinLocus CoeruleusGerbillinaeSomatostatinhormones hormone substitutes and hormone antagonistsNeurotensinNaunyn-Schmiedeberg's archives of pharmacology
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Effect of acidosis on lipid peroxidation in brain slices.

1991

Acidification of the incubation medium markedly increased lipid peroxidation of cortical brain slices. Lactic acidosis caused a more extensive lipid peroxidation than did phosphoric acidosis (+35% at pH 6 and +81% at pH 5), probably due to the rapid diffusion of the protonated form of lactic acid across cell membranes. These results support the hypothesis that free radical mechanisms may be involved in the cytotoxicity of acidosis.

Malemedicine.medical_specialtyIn Vitro TechniquesPhosphatesLipid peroxidationchemistry.chemical_compoundSlice preparationInternal medicinemedicineAnimalsCytotoxicityMolecular BiologyIncubationAcidosisCerebral CortexChemistryGeneral NeuroscienceBrainRats Inbred StrainsMetabolismHydrogen-Ion Concentrationmedicine.diseaseLactic acidRatsKineticsEndocrinologyBiochemistryLactic acidosisLactatesNeurology (clinical)Lipid Peroxidationmedicine.symptomAcidosisDevelopmental BiologyBrain research
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Temperature controller for brain slice chambers using a current sink output stage.

1985

Temperature controlMaterials scienceBiomedical EngineeringTemperatureBrainHuman physiologyEquipment DesignThermostatHippocampusComputer Science Applicationslaw.inventionRatsSlice preparationlawControl theoryAnimalsStage (hydrology)Current (fluid)Sink (computing)Medicalbiological engineeringcomputing
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